acarbose

Brand: Precose

Prototype Drug
Drug Class: alpha-glucosidase inhibitor
Drug Family: antidiabetic
Subclass: intestinal alpha-glucosidase inhibitor
Organ Systems: endocrine

Mechanism of Action

Competitively inhibits intestinal alpha-glucosidases (maltase, sucrase, isomaltase, glucoamylase) at the brush border of the small intestine, slowing the breakdown of complex carbohydrates and sucrose to glucose. This delays and reduces postprandial glucose absorption.

alpha-glucosidase enzymes (intestinal brush border)

Indications

  • type 2 diabetes mellitus (reduces postprandial hyperglycemia)

Contraindications

  • inflammatory bowel disease
  • colonic ulceration
  • intestinal obstruction
  • cirrhosis
  • severe renal impairment (CrCl <25 mL/min)

Adverse Effects

Common

  • flatulence (most common — up to 77%)
  • diarrhea
  • abdominal cramping

Serious

  • hepatotoxicity (rare, with high doses)
  • ileus (rare)

Pharmacokinetics (ADME)

Absorption oral; minimally absorbed (<2%); acts locally in GI tract
Distribution local GI tract
Metabolism metabolized by intestinal bacteria; minor hepatic metabolism of absorbed fraction
Excretion fecal (primary); minor renal excretion of absorbed fraction
Half-life 2 hours
Onset rapid (during meal)
Peak during meal
Duration postprandial period
Protein Binding minimal
Vd minimal

Drug Interactions

Drug / Agent Mechanism Severity
digestive enzymes (amylase, pancreatin) antagonize action of acarbose moderate
insulin or sulfonylureas acarbose combined with secretagogues can increase hypoglycemia risk; treat hypoglycemia with GLUCOSE (not sucrose) moderate

Nursing Considerations

  1. Administer with the FIRST BITE of each main meal (3 times daily); taking before or after a meal reduces efficacy.
  2. If hypoglycemia occurs while on acarbose (in combination with insulin/SU), treat ONLY with pure glucose (dextrose or glucose tabs) NOT sucrose (sucrose cannot be digested when acarbose is active).
  3. Monitor LFTs at 3-month intervals for first year; rare hepatotoxicity at high doses.
  4. GI side effects are dose-related; start at low dose (25 mg) and titrate slowly; side effects typically decrease over time as gut flora adapts.

Clinical Pearls

  • The critical clinical pearl: hypoglycemia occurring in a patient on acarbose MUST be treated with glucose tablets or IV dextrose — table sugar (sucrose) cannot be properly digested because sucrase is inhibited.
  • Acarbose has a favorable safety profile (minimal systemic absorption) but is limited in clinical use by its GI side effects; it is more commonly used in Asia than in the United States.

Safety Profile

Pregnancy insufficient-data
Lactation avoid
Renal Adjustment Required
Hepatic Adjustment Required
TDM Not required

Concordance Terms

Cross-referenced clinical concepts — click any term to see all content where it appears.