alosetron

Brand: Lotronex

⚠ BBW Prototype: ondansetron
Drug Class: 5-HT3 receptor antagonist (GI-selective)
Drug Family: GI agent
Subclass: selective 5-HT3 antagonist for IBS
Organ Systems: gastrointestinal

Mechanism of Action

Selectively antagonizes 5-HT3 receptors in the enteric nervous system and on visceral afferent neurons. This slows colonic transit, reduces intestinal secretion, and decreases visceral hypersensitivity, addressing both the diarrhea and pain components of IBS-D.

5-HT3 receptors (enteric nervous system and visceral afferents)

Indications

  • severe diarrhea-predominant IBS (IBS-D) in women who have not responded to conventional therapy

Contraindications

  • IBS with constipation
  • chronic constipation
  • Crohn's disease
  • ulcerative colitis
  • ischemic colitis history
  • hypercoagulable state
  • severe hepatic impairment

Adverse Effects

Common

  • constipation (up to 29%)
  • abdominal pain

Serious

  • ischemic colitis
  • severe constipation requiring hospitalization or surgery

Pharmacokinetics (ADME)

Absorption oral bioavailability ~50–60%; affected by gender (women have higher exposure)
Distribution 82% protein bound
Metabolism hepatic via CYP1A2, CYP3A4, CYP2C9
Excretion renal (~73%)
Half-life 1.5 hours
Onset days
Peak 1 hour
Duration continuous dosing
Protein Binding 82%
Vd 65–95 L

Drug Interactions

Drug / Agent Mechanism Severity
fluvoxamine CYP1A2 inhibition increases alosetron exposure 6-fold; contraindicated contraindicated
strong CYP1A2 inhibitors markedly increased alosetron levels major

Nursing Considerations

  1. Only prescribers enrolled in the Lotronex REMS program may prescribe alosetron; verify enrollment status.
  2. Discontinue immediately at first signs of ischemic colitis (rectal bleeding, bloody diarrhea, new or worsening abdominal pain); do not reinstate.
  3. Instruct patient to stop use if constipation develops and resume only when constipation resolves at the lowest dose.
  4. Approved only for women with severe IBS-D unresponsive to conventional therapy — confirm severity and refractoriness before prescribing.

Clinical Pearls

  • Alosetron was voluntarily withdrawn from the market in 2000 due to serious GI adverse events and re-introduced in 2002 under a REMS program with restricted prescribing — it is one of the few drugs reintroduced under a formal risk management program.
  • Its selectivity for 5-HT3 receptors in the enteric nervous system rather than the CTZ distinguishes its GI-targeted effects from antiemetic 5-HT3 antagonists.

Safety Profile

Pregnancy use-with-caution
Lactation avoid
Renal Adjustment Not required
Hepatic Adjustment Required
TDM Not required

Concordance Terms

Cross-referenced clinical concepts — click any term to see all content where it appears.