alteplase

Brand: Activase, Cathflo Activase

⚠ BBW ISMP High Alert Prototype Drug
Drug Class: thrombolytic agent
Drug Family: thrombolytic
Subclass: recombinant tissue plasminogen activator (tPA)
Organ Systems: cardiovascularhematology-oncology

Mechanism of Action

Recombinant human tPA that preferentially binds fibrin within a thrombus and converts plasminogen to plasmin, causing clot dissolution; fibrin binding concentrates thrombolytic activity at the clot site, minimizing systemic plasminogen activation.

plasminogen (fibrin-bound)fibrin clot

Indications

  • acute ischemic stroke (within 3-4.5 hours of symptom onset)
  • acute STEMI
  • massive pulmonary embolism
  • occluded central venous access devices (Cathflo)

Contraindications

  • active internal bleeding
  • recent (within 3 months) intracranial or intraspinal surgery or trauma
  • history of intracranial hemorrhage
  • intracranial neoplasm
  • AV malformation
  • severe uncontrolled hypertension
  • suspected aortic dissection

Adverse Effects

Common

  • bleeding at puncture sites
  • ecchymosis
  • epistaxis

Serious

  • intracranial hemorrhage
  • major systemic bleeding
  • angioedema (with ACE inhibitor use)
  • embolic events (cholesterol embolism)

Pharmacokinetics (ADME)

Absorption IV administration only
Distribution rapidly distributed; Vd approximately 4-8 L (blood volume)
Metabolism rapidly cleared by the liver; endocytosis via LRP1 receptor
Excretion hepatic metabolism; minimal renal excretion
Half-life approximately 5 minutes (initial); approximately 26 minutes (terminal)
Onset immediate (IV)
Peak end of infusion
Duration fibrinolysis continues for up to 1 hour after infusion
Protein Binding low
Vd 4-8 L

Drug Interactions

Drug / Agent Mechanism Severity
anticoagulants (heparin, warfarin, DOACs) increased bleeding risk when used concurrently or before/after thrombolysis major
antiplatelet agents (aspirin, clopidogrel) additive risk of intracranial and systemic hemorrhage major
ACE inhibitors angioedema risk increased with alteplase; mechanism involves bradykinin moderate

Nursing Considerations

  1. Obtain IV access in non-compressible sites before alteplase administration; avoid arterial punctures during and for 24 hours after infusion; apply pressure for at least 30 minutes to any compressible bleeding site.
  2. Monitor neurological status every 15 minutes during infusion and every 30 minutes for 6 hours post-infusion; any decline in neurological status, new headache, nausea, or vomiting suggests intracranial hemorrhage — STOP infusion and notify physician immediately.
  3. Hold heparin during alteplase infusion for STEMI; do not administer aspirin or anticoagulants for 24 hours post-infusion in stroke patients without imaging confirmation of no hemorrhage.
  4. Strict blood pressure management is essential in stroke: maintain BP below 185/110 mmHg before and during alteplase infusion; use IV labetalol or nicardipine per protocol.

Clinical Pearls

  • For acute ischemic stroke, the therapeutic window extends to 4.5 hours from symptom onset in eligible patients; time is brain — every 30-minute delay to treatment reduces the number of patients who regain independent function.
  • Alteplase doses differ significantly by indication: 0.9 mg/kg (max 90 mg) for stroke, 100 mg over 2 hours for massive PE — nursing staff must confirm the indication and correct dosing regimen before administration.

Safety Profile

Pregnancy use-with-caution
Lactation insufficient-data
Renal Adjustment Not required
Hepatic Adjustment Not required
TDM Not required