amikacin

Brand: Amikin

⚠ BBW TDM Required Prototype: gentamicin
Drug Class: aminoglycoside antibiotic
Drug Family: antibiotic
Subclass: broad-spectrum aminoglycoside resistant to most aminoglycoside-modifying enzymes
Organ Systems: infectious-disease

Mechanism of Action

Binds 30S ribosomal subunit causing misreading and inhibition of translocation; amikacin's chemical structure confers resistance to most aminoglycoside-inactivating enzymes, making it the aminoglycoside of last resort for resistant gram-negative infections.

30S ribosomal subunit

Indications

  • serious gram-negative infections resistant to gentamicin and tobramycin
  • hospital-acquired pneumonia and VAP (gram-negative)
  • urinary tract infections (serious)
  • NTM infections (Mycobacterium abscessus)
  • combination therapy for MDR Acinetobacter baumannii

Contraindications

  • aminoglycoside hypersensitivity
  • pregnancy (ototoxicity risk)

Adverse Effects

Common

  • nephrotoxicity
  • ototoxicity (vestibular and cochlear)
  • elevated BUN/creatinine

Serious

  • irreversible hearing loss
  • vestibular toxicity (vertigo, nystagmus)
  • acute kidney injury
  • neuromuscular blockade

Pharmacokinetics (ADME)

Absorption IV/IM only
Distribution extracellular; poor CNS penetration
Metabolism not metabolized
Excretion renal (unchanged)
Half-life 2–3 hours (normal renal function)
Onset immediate (IV)
Peak 30–90 min post-infusion
Duration 8–24 hours
Protein Binding 0–10%
Vd low

Drug Interactions

Drug / Agent Mechanism Severity
loop diuretics additive ototoxicity major
vancomycin/colistin additive nephrotoxicity major
neuromuscular blocking agents enhanced neuromuscular blockade; respiratory depression major

Nursing Considerations

  1. Monitor amikacin peaks (goal 20–35 mcg/mL for traditional dosing or 56–64 mcg/mL for extended-interval) and troughs (<5 mcg/mL traditional; undetectable extended-interval) — obtain timing precisely.
  2. Monitor serial BUN, SCr, and urine output; assess for ototoxicity with audiometry and vestibular testing when therapy extends beyond 7 days.
  3. Avoid concurrent nephrotoxic agents; if loop diuretics are required, separate dosing times and monitor closely.
  4. Extended-interval (high-dose once-daily) dosing maximizes peak-dependent killing and may reduce nephrotoxicity; verify pharmacy-determined dosing strategy.

Clinical Pearls

  • Amikacin is the aminoglycoside least susceptible to enzymatic inactivation, because its hydroxyl and amino groups that are targets of aminoglycoside-modifying enzymes are protected by hydroxyl-butanoyl substitution.
  • In the context of MDR and XDR gram-negative infections, amikacin is often the only aminoglycoside with in vitro activity, though carbapenem-resistant organisms require combination regimens.

Safety Profile

Pregnancy use-with-caution
Lactation use-with-caution
Renal Adjustment Required
Hepatic Adjustment Not required
TDM Required