atovaquone-proguanil

Brand: Malarone

Prototype Drug
Drug Class: combination antimalarial
Drug Family: antiparasitic
Subclass: mitochondrial complex III inhibitor + antifolate combination
Organ Systems: infectious-disease

Mechanism of Action

Atovaquone inhibits the mitochondrial electron transport chain at cytochrome bc1 complex, collapsing the mitochondrial membrane potential; cycloguanil (active metabolite of proguanil) inhibits dihydrofolate reductase; combination is synergistic and active against both liver and blood stages.

mitochondrial cytochrome bc1 complex III (atovaquone)dihydrofolate reductase (cycloguanil, proguanil metabolite)

Indications

  • malaria prophylaxis for travelers to chloroquine-resistant regions
  • malaria treatment (P. falciparum including chloroquine-resistant)

Contraindications

  • atovaquone/proguanil hypersensitivity
  • severe renal impairment (CrCl <30 mL/min — use alternative for prophylaxis)
  • pregnancy (insufficient data)

Adverse Effects

Common

  • GI upset
  • headache
  • abdominal pain

Serious

  • hepatitis (rare)
  • Stevens-Johnson syndrome (rare)
  • neutropenia (rare)

Pharmacokinetics (ADME)

Absorption atovaquone: 40% bioavailability fasting, significantly increased (2.5–3x) with fatty food; proguanil: ~55% bioavailability; administer with food or milky drink
Distribution atovaquone: large Vd, highly lipophilic; proguanil/cycloguanil: moderate distribution
Metabolism atovaquone: minimal; proguanil metabolized by CYP2C19 to active cycloguanil
Excretion atovaquone: fecal (>94% unchanged); proguanil/cycloguanil: renal
Half-life atovaquone 2–3 days; proguanil 12–21 hours
Onset rapid
Peak 2–4 hours
Duration once-daily dosing
Protein Binding atovaquone 99%; proguanil 75%
Vd large (atovaquone)

Drug Interactions

Drug / Agent Mechanism Severity
rifampin reduces atovaquone levels by ~50%; avoid combination major
warfarin atovaquone may enhance anticoagulant effect; monitor INR moderate
metoclopramide reduces atovaquone absorption; avoid combination moderate

Nursing Considerations

  1. Must be taken with food or milky drink — atovaquone absorption is critically dependent on fat content.
  2. For prophylaxis: start 1–2 days before travel and continue for 7 days after return (shorter post-travel course than chloroquine or mefloquine).
  3. Advise patients to take at the same time each day; if vomiting occurs within 1 hour of dose, repeat the dose.
  4. Atovaquone-proguanil is NOT effective for P. vivax hypnozoites; primaquine or tafenoquine must be added if P. vivax exposure is possible.

Clinical Pearls

  • Atovaquone-proguanil is one of three recommended options for malaria chemoprophylaxis in chloroquine-resistant areas (along with mefloquine and doxycycline); its advantages include once-daily dosing, minimal neuropsychiatric side effects compared to mefloquine, and only 7 days post-travel continuation.
  • CYP2C19 poor metabolizers produce less cycloguanil from proguanil, theoretically reducing antifolate efficacy; however, atovaquone alone provides substantial antimalarial activity, and clinical failures attributable solely to CYP2C19 status are uncommon.

Safety Profile

Pregnancy use-with-caution
Lactation use-with-caution
Renal Adjustment Required
Hepatic Adjustment Not required
TDM Not required