codeine

Brand: Tylenol with Codeine, Robitussin AC (with guaifenesin)

⚠ BBW ISMP High Alert Prototype: morphine
Drug Class: opioid analgesic and antitussive
Drug Family: opioid
Subclass: natural opioid prodrug
Organ Systems: cns

Mechanism of Action

Prodrug requiring CYP2D6-mediated conversion to morphine for analgesic activity; approximately 10% of codeine is converted to morphine. Inhibits cough reflex via central mechanisms. Ultrarapid CYP2D6 metabolizers produce dangerously high morphine levels; poor metabolizers receive no analgesic benefit.

mu-opioid receptor (MOR, via morphine metabolite)

Indications

  • mild to moderate pain
  • antitussive (cough suppression)

Contraindications

  • children <12 years old (FDA safety communication 2013)
  • post-tonsillectomy/adenoidectomy pain in children
  • ultrarapid CYP2D6 metabolizers (relative)
  • breastfeeding (risk of infant death from morphine in milk of ultrarapid metabolizer mothers)

Adverse Effects

Common

  • constipation
  • nausea
  • sedation
  • dizziness

Serious

  • respiratory depression (especially in ultrarapid CYP2D6 metabolizers)
  • death in pediatric patients
  • dependence and addiction

Pharmacokinetics (ADME)

Absorption well absorbed orally; bioavailability ~90%
Distribution protein binding ~7-25%
Metabolism CYP2D6 converts ~10% to morphine (active); CYP3A4 converts ~80% to norcodeine (inactive); glucuronidation produces codeine-6-glucuronide
Excretion primarily renal
Half-life 3-4 hours
Onset 30-45 minutes
Peak 1-1.5 hours
Duration 4-6 hours
Protein Binding 7-25%
Vd 3-4 L/kg

Drug Interactions

Drug / Agent Mechanism Severity
CYP2D6 inhibitors (fluoxetine, paroxetine, bupropion) block conversion to morphine; reduce analgesic efficacy moderate
CNS depressants additive respiratory and CNS depression major

Nursing Considerations

  1. Codeine is contraindicated in patients under 18 years for cough/cold (FDA 2017) and under 12 years for pain; verify patient age before administration and clarify orders for pediatric patients.
  2. Breastfeeding mothers who are CYP2D6 ultrarapid metabolizers (prevalence 1-7% in most populations, up to 29% in North African populations) produce milk with high morphine concentrations; monitor breastfed infants for respiratory depression.
  3. The variability in codeine response based on CYP2D6 genotype (from no effect to life-threatening toxicity) makes it one of the clearest examples of pharmacogenomics influencing clinical practice.
  4. A combination product with acetaminophen (Tylenol #3, #4) requires verification that total acetaminophen dose from all sources does not exceed safe limits.

Clinical Pearls

  • Codeine's 'prodrug' status means that its entire analgesic effect depends on CYP2D6 conversion to morphine; poor metabolizers (7-10% of Caucasians) receive essentially no analgesia, while ultrarapid metabolizers receive dangerous morphine levels.
  • Several deaths in pediatric patients after tonsillectomy led to codeine's pediatric restriction; the deaths occurred in children who were CYP2D6 ultrarapid metabolizers — a pharmacogenomic disaster that changed FDA labeling.

Safety Profile

Pregnancy avoid
Lactation avoid
Renal Adjustment Required
Hepatic Adjustment Required
TDM Not required

Concordance Terms

Cross-referenced clinical concepts — click any term to see all content where it appears.