doxorubicin

Brand: Adriamycin, Doxil (liposomal)

⚠ BBW ISMP High Alert Prototype Drug
Drug Class: anthracycline antineoplastic antibiotic
Drug Family: antineoplastic
Subclass: prototype anthracycline
Organ Systems: hematology-oncology

Mechanism of Action

Intercalates into DNA, blocking topoisomerase II from resolving DNA strand breaks; generates reactive oxygen species via redox cycling; intercalation and free radical mechanisms contribute to DNA damage and apoptosis; cardiotoxicity results from iron-mediated ROS generation in cardiac mitochondria.

topoisomerase IIDNA intercalationreactive oxygen species generation

Indications

  • breast cancer (adjuvant and metastatic)
  • acute leukemias
  • Hodgkin and non-Hodgkin lymphoma
  • soft tissue sarcoma
  • bladder, ovarian, thyroid cancers
  • Kaposi sarcoma (liposomal formulation)

Contraindications

  • doxorubicin hypersensitivity
  • severe myocardial insufficiency
  • previous total anthracycline dose near lifetime maximum
  • recent MI or severe cardiac arrhythmia

Adverse Effects

Common

  • myelosuppression (dose-limiting)
  • alopecia (complete)
  • nausea/vomiting
  • red urine (not hematuria — normal discoloration from drug)
  • mucositis

Serious

  • cardiomyopathy (dose-dependent, cumulative, potentially irreversible)
  • acute cardiotoxicity (arrhythmias during infusion)
  • myelosuppression
  • extravasation necrosis (vesicant — severe)
  • secondary leukemia (AML)
  • radiation recall

Pharmacokinetics (ADME)

Absorption IV only (significant first-pass inactivation if oral)
Distribution large Vd; concentrates in tissues; limited CNS penetration
Metabolism hepatic; active metabolite doxorubicinol (cardiotoxic)
Excretion biliary/fecal (primary); renal minor; dose reduction required in hepatic impairment
Half-life 20–48 hours (terminal)
Onset immediate (IV)
Peak end of infusion
Duration 3-week cycles
Protein Binding 75%
Vd very large

Drug Interactions

Drug / Agent Mechanism Severity
trastuzumab both cardiotoxic; additive cardiomyopathy risk — avoid concurrent use; sequence trastuzumab after anthracycline major
cyclosporine increases doxorubicin levels and toxicity via P-gp inhibition major
paclitaxel sequence-dependent interaction; paclitaxel after doxorubicin increases cardiotoxicity major

Nursing Considerations

  1. Doxorubicin is a potent vesicant — peripheral IV access must be checked for patency before EACH cycle; extravasation causes severe, progressive tissue necrosis requiring surgical debridement.
  2. Obtain echocardiogram or MUGA scan to assess LVEF before initiating therapy, after cumulative doses of 250–300 mg/m², and before each additional dose once cumulative dose exceeds 400 mg/m².
  3. Warn patients that urine will appear red-orange for 1–2 days after treatment — reassure them this is the drug, not blood.
  4. Cumulative lifetime dose is typically limited to 550 mg/m² (450 mg/m² with prior mediastinal radiation) — communicate this limit across providers.

Clinical Pearls

  • Doxorubicin's cardiomyopathy is mediated by iron-catalyzed free radical production in cardiac mitochondria (which lack catalase); dexrazoxane, an iron chelator, is FDA-approved to prevent doxorubicin cardiomyopathy by blocking iron-mediated ROS generation in the heart.
  • The liposomal formulation (Doxil) achieves preferential tumor accumulation via the enhanced permeability and retention (EPR) effect while reducing cardiac exposure, significantly lowering cardiomyopathy risk.

Safety Profile

Pregnancy contraindicated
Lactation avoid
Renal Adjustment Not required
Hepatic Adjustment Required
TDM Not required