empagliflozin

Brand: Jardiance

Prototype Drug
Drug Class: antidiabetic
Drug Family: antidiabetic
Subclass: SGLT2 inhibitor
Organ Systems: endocrinecardiovascularrenal

Mechanism of Action

Inhibits SGLT2 in the proximal renal tubule, reducing renal glucose reabsorption and causing glucosuria; glucose-lowering is insulin-independent; also reduces tubular sodium reabsorption (natriuresis), reducing preload and afterload, and has direct cardiac and renal protective effects beyond glycemic control.

sodium-glucose cotransporter 2 (SGLT2)

Indications

  • type 2 diabetes mellitus (glycemic control + CV risk reduction)
  • heart failure with reduced ejection fraction (HFrEF) — reduces HF hospitalization and CV death
  • chronic kidney disease (CKD) — reduces progression
  • off-label: heart failure with preserved ejection fraction (HFpEF)

Contraindications

  • eGFR <20 mL/min (for CV/HF benefit; no longer specified for T2DM glycemic indication)
  • active genital mycotic infections
  • type 1 diabetes (risk of DKA without significant hyperglycemia — euglycemic DKA)

Adverse Effects

Common

  • genital mycotic infections (yeast infections — most common in women)
  • urinary tract infections
  • polyuria
  • dehydration/orthostatic hypotension

Serious

  • euglycemic diabetic ketoacidosis (DKA) — blood glucose may be <250 mg/dL; high anion gap metabolic acidosis
  • lower limb amputations (canagliflozin > empagliflozin)
  • Fournier's gangrene (necrotizing fasciitis of perineum — rare)
  • acute kidney injury (volume depletion)
  • urosepsis and pyelonephritis

Pharmacokinetics (ADME)

Absorption ~86% oral bioavailability; take with or without food
Distribution Protein binding 86%; Vd ~73 L
Metabolism UGT2B7 and UGT1A3 (glucuronidation); minimal CYP involvement
Excretion Renal (~41% unchanged) and fecal (~41%)
Half-life ~12 hours
Onset 24 hours for glucose lowering; weeks for CV/renal effects
Peak 1.5 hours
Duration 24 hours
Protein Binding 86%
Vd ~73 L

Drug Interactions

Drug / Agent Mechanism Severity
insulin / insulin secretagogues (sulfonylureas) additive hypoglycemia; reduce insulin/sulfonylurea dose when adding SGLT2i moderate
diuretics additive volume depletion and hypotension risk moderate

Nursing Considerations

  1. Counsel patients about genital mycotic infections (most common in women with history of candidiasis) — maintain genital hygiene; may require antifungal treatment; drug holiday if recurrent
  2. Hold SGLT2 inhibitors 3–4 days before surgery or major procedures due to euglycemic DKA risk in fasting patients on insulin; instruct patient to report nausea, vomiting, abdominal pain, and difficult breathing (DKA symptoms)
  3. Monitor for signs of UTI; obtain urine culture and treat promptly; SGLT2 inhibitors cause glucosuria which predisposes to urinary tract infections
  4. For HF indication (Jardiance): empagliflozin demonstrated 25% relative risk reduction in CV death/HF hospitalization in EMPA-REG OUTCOME trial; now standard of care in HFrEF per AHA/ACC guidelines

Clinical Pearls

  • Euglycemic DKA is the most dangerous and underrecognized SGLT2 inhibitor complication — patients present with DKA symptoms but blood glucose may be only mildly elevated (200–250 mg/dL), leading to missed or delayed diagnosis
  • SGLT2 inhibitors have emerged as the most impactful drug class in cardiology and nephrology this decade — they reduce HF hospitalization, CV death, and CKD progression through mechanisms beyond glycemic control (osmotic diuresis, tubuloglomerular feedback, anti-inflammatory effects)

Safety Profile

Pregnancy avoid
Lactation avoid
Renal Adjustment Required
Hepatic Adjustment Not required
TDM Not required
Guideline Update pending