fludrocortisone acetate

Brand: Florinef

Prototype Drug
Drug Class: mineralocorticoid
Drug Family: corticosteroid
Subclass: synthetic mineralocorticoid
Organ Systems: endocrine

Mechanism of Action

Potent synthetic mineralocorticoid that binds the mineralocorticoid receptor in renal collecting duct principal cells, stimulating expression of sodium channels (ENaC) and Na+/K+-ATPase pumps, causing sodium and water retention with potassium and hydrogen ion secretion.

mineralocorticoid receptor (MR) in renal collecting duct

Indications

  • primary adrenal insufficiency (Addison's disease) — mineralocorticoid replacement
  • salt-losing congenital adrenal hyperplasia
  • orthostatic hypotension (off-label)

Contraindications

  • systemic fungal infections
  • hypertension
  • heart failure
  • edema states

Adverse Effects

Common

  • edema
  • hypertension
  • weight gain
  • headache

Serious

  • hypokalemia
  • hypertension (severe)
  • congestive heart failure
  • Cushing's syndrome (at high doses)

Pharmacokinetics (ADME)

Absorption oral; rapid and complete absorption
Distribution ~42% protein bound
Metabolism hepatic
Excretion renal
Half-life 3.5 hours (plasma); 18–36 hours (biologic effect)
Onset hours
Peak 1.7 hours
Duration 24–36 hours (biologic)
Protein Binding 42%
Vd moderate

Drug Interactions

Drug / Agent Mechanism Severity
diuretics additive electrolyte effects; thiazides may potentiate hypokalemia moderate
NSAIDs blunt mineralocorticoid-mediated sodium retention, reducing fludrocortisone efficacy moderate
potassium-depleting agents additive hypokalemia moderate

Nursing Considerations

  1. Monitor blood pressure, electrolytes (sodium, potassium), and weight at each visit during dose titration.
  2. Typical dose for adrenal insufficiency is 0.05–0.1 mg daily; titrate to normalize electrolytes and blood pressure.
  3. Educate patients on sick day dosing for hydrocortisone (not fludrocortisone; the glucocorticoid dose is increased during illness).
  4. Warn patients to avoid excessive sodium restriction, which could counteract mineralocorticoid replacement.

Clinical Pearls

  • Fludrocortisone is the only oral mineralocorticoid available; dexamethasone and prednisone have negligible mineralocorticoid activity and cannot substitute for fludrocortisone in adrenal insufficiency.
  • Adequate mineralocorticoid replacement (avoiding hyponatremia and hyperkalemia) is essential for hemodynamic stability in Addison's disease — insufficient fludrocortisone dosing contributes to orthostatic hypotension and salt-wasting crisis.

Safety Profile

Pregnancy use-with-caution
Lactation use-with-caution
Renal Adjustment Not required
Hepatic Adjustment Not required
TDM Not required