fluticasone furoate

Brand: Arnuity Ellipta (ICS alone), Breo Ellipta (with vilanterol), Trelegy Ellipta (with vilanterol + umeclidinium)

Prototype: fluticasone propionate
Drug Class: inhaled corticosteroid (ICS)
Drug Family: corticosteroid
Subclass: synthetic glucocorticoid — once-daily ICS
Organ Systems: respiratory

Mechanism of Action

Synthetic glucocorticoid with high glucocorticoid receptor affinity (approximately 29-fold greater than fluticasone propionate for GR); binds intracellular GR, translocates to the nucleus, transactivates anti-inflammatory genes and transrepresses pro-inflammatory transcription factors (NF-kB, AP-1), reducing airway eosinophilic inflammation and cytokine production.

glucocorticoid receptor (GR)airway inflammatory cells

Indications

  • asthma maintenance therapy
  • COPD with frequent exacerbations (in combination products)

Contraindications

  • primary treatment of acute bronchospasm
  • hypersensitivity to fluticasone furoate or milk proteins

Adverse Effects

Common

  • oropharyngeal candidiasis
  • nasopharyngitis
  • upper respiratory infection
  • headache
  • dysphonia

Serious

  • adrenal suppression (high doses, prolonged use)
  • systemic corticosteroid effects (osteoporosis, cataracts, growth suppression in children)
  • increased pneumonia risk in COPD patients

Pharmacokinetics (ADME)

Absorption inhaled; Cmax within 0.5-1 hour; absolute systemic bioavailability approximately 15%
Distribution Vd approximately 661 L; 99.7% protein bound
Metabolism extensive hepatic CYP3A4 first-pass metabolism; rapidly inactivated to inactive carboxylic acid metabolite
Excretion fecal (~101%) — enterohepatic cycling; renal minimal
Half-life approximately 24 hours
Onset anti-inflammatory effect within days; maximal benefit over weeks
Peak 0.5-1 hour (systemic)
Duration 24 hours (once-daily dosing)
Protein Binding 99.7%
Vd approximately 661 L

Drug Interactions

Drug / Agent Mechanism Severity
strong CYP3A4 inhibitors (ketoconazole, ritonavir) increase systemic fluticasone furoate exposure; risk of adrenal suppression and systemic corticosteroid effects major
other inhaled corticosteroids additive systemic corticosteroid exposure if combined; avoid unless intentional moderate

Nursing Considerations

  1. Instruct patients to rinse mouth with water and spit immediately after each inhalation to prevent oropharyngeal candidiasis; inspect mouth at each visit for white plaques.
  2. Fluticasone furoate is once-daily, unlike fluticasone propionate which is twice-daily; verify the formulation on the prescription and confirm the patient understands the correct dosing frequency.
  3. Monitor for systemic corticosteroid effects with prolonged high-dose use: assess for cushingoid features, bone density (particularly in postmenopausal women), and screen for cataracts and glaucoma annually.
  4. Do not use for acute asthma exacerbations; instruct patients that slow onset makes ICS unsuitable for rescue use; a SABA must be prescribed alongside for acute symptom relief.

Clinical Pearls

  • Fluticasone furoate's superior glucocorticoid receptor binding affinity compared with fluticasone propionate supports once-daily dosing rather than twice-daily, but no head-to-head trial has shown clinical superiority over fluticasone propionate at equivalent doses.
  • In COPD patients, ICS therapy including fluticasone furoate is associated with increased pneumonia risk; the ACC/AHA and GOLD guidelines recommend reserving ICS use for COPD patients with frequent exacerbations and blood eosinophil counts above 100-300 cells/mcL.

Safety Profile

Pregnancy use-with-caution
Lactation insufficient-data
Renal Adjustment Not required
Hepatic Adjustment Required
TDM Not required

Concordance Terms

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