itraconazole

Brand: Sporanox, Onmel

⚠ BBW Beers Criteria TDM Required Prototype: fluconazole
Drug Class: triazole antifungal
Drug Family: antifungal
Subclass: first-generation triazole with broad spectrum
Organ Systems: infectious-disease

Mechanism of Action

Inhibits fungal CYP51, blocking ergosterol synthesis and disrupting fungal cell membrane integrity; broader spectrum than fluconazole, including Aspergillus; also a potent CYP3A4 and P-gp inhibitor.

fungal CYP51 (lanosterol 14-alpha-demethylase)

Indications

  • aspergillosis (prophylaxis and treatment)
  • histoplasmosis
  • blastomycosis
  • sporotrichosis
  • onychomycosis
  • paracoccidioidomycosis

Contraindications

  • itraconazole hypersensitivity
  • concomitant use of CYP3A4-cleared drugs with narrow therapeutic index (pimozide, cisapride, lovastatin, simvastatin, ergotamine, quinidine)
  • negative inotropic effect — avoid in ventricular dysfunction

Adverse Effects

Common

  • nausea
  • vomiting
  • abdominal pain
  • headache

Serious

  • hepatotoxicity
  • negative inotropy/heart failure exacerbation
  • severe drug interactions via CYP3A4 and P-gp inhibition
  • peripheral neuropathy (prolonged use)

Pharmacokinetics (ADME)

Absorption highly variable; capsules require food and gastric acid; solution taken on empty stomach for better systemic absorption; avoid with acid-suppressing drugs
Distribution large Vd; extensively distributed to tissues; plasma levels may not reflect tissue concentrations
Metabolism extensive CYP3A4 metabolism; active metabolite hydroxyitraconazole (equal antifungal potency); also a strong CYP3A4 and P-gp inhibitor
Excretion primarily fecal (54% as metabolites); renal <1% unchanged
Half-life 40 hours (multiple dose)
Onset variable
Peak 1–5 hours (capsules with food)
Duration 24 hours
Protein Binding 99.8%
Vd very large (11 L/kg)

Drug Interactions

Drug / Agent Mechanism Severity
simvastatin/lovastatin CYP3A4 inhibition raises statin levels dramatically; rhabdomyolysis risk — contraindicated major
warfarin CYP3A4 inhibition increases INR; monitor closely major
tacrolimus/cyclosporine CYP3A4 inhibition markedly increases immunosuppressant levels; require dose adjustment major
calcium channel blockers CYP3A4 inhibition plus additive negative inotropy; monitor for edema and hypotension major

Nursing Considerations

  1. Capsules must be taken with food and in an acidic environment; do not administer capsules with antacids or acid-suppressing drugs — instruct patient to take with a cola beverage if on acid suppression.
  2. Itraconazole oral solution should be taken on an empty stomach for better systemic absorption (opposite of capsules).
  3. Screen all medications for CYP3A4-based drug interactions before initiating; the interaction list is extensive.
  4. Monitor LFTs monthly; hepatotoxicity can be severe — stop drug and notify prescriber if LFTs rise >3× ULN.

Clinical Pearls

  • Itraconazole's active metabolite (hydroxyitraconazole) has antifungal activity equal to the parent drug, contributing to its efficacy, but both strongly inhibit CYP3A4 and P-gp, creating a vast drug interaction burden.
  • For histoplasmosis and blastomycosis, itraconazole is the drug of choice for non-severe disease; the solution formulation achieves more reliable bioavailability than capsules.

Safety Profile

Pregnancy contraindicated
Lactation avoid
Renal Adjustment Not required
Hepatic Adjustment Required
TDM Required

Concordance Terms

Cross-referenced clinical concepts — click any term to see all content where it appears.