lacosamide

Brand: Vimpat

Prototype Drug
Drug Class: antiepileptic drug
Drug Family: antiepileptic
Subclass: selective sodium channel slow inactivation enhancer
Organ Systems: cns

Mechanism of Action

Novel mechanism of sodium channel modulation: selectively enhances slow inactivation of voltage-gated Na+ channels (vs. fast inactivation targeted by phenytoin, carbamazepine) and binds CRMP-2 (collapsin response mediator protein-2), reducing neuronal hyperexcitability in a manner complementary to other Na+ channel blockers.

voltage-gated sodium channels (selective enhancement of slow inactivation)

Indications

  • partial (focal) seizures (adjunctive or monotherapy in adults)
  • primary generalized tonic-clonic seizures (adjunctive)

Contraindications

  • hypersensitivity to lacosamide

Adverse Effects

Common

  • diplopia
  • headache
  • dizziness
  • nausea
  • ataxia

Serious

  • PR interval prolongation (cardiac conduction); monitor ECG
  • cardiac arrhythmias (in patients with cardiac conduction disorders)
  • suicidal ideation (class AED effect)
  • drug reaction with eosinophilia and systemic symptoms (DRESS)

Pharmacokinetics (ADME)

Absorption oral or IV; bioavailability ~100%
Distribution <15% protein bound
Metabolism hepatic via CYP2C19 (minor); mainly excreted unchanged
Excretion renal (95%)
Half-life 13 hours
Onset rapid
Peak 1–4 hours
Duration 12 hours (BID dosing)
Protein Binding <15%
Vd 0.6 L/kg

Drug Interactions

Drug / Agent Mechanism Severity
drugs prolonging PR interval (carbamazepine, lidocaine, beta-blockers) additive PR prolongation; risk of AV block moderate

Nursing Considerations

  1. Obtain baseline ECG; lacosamide prolongs PR interval; use with caution in patients with cardiac conduction disorders or on other PR-prolonging drugs.
  2. IV form available for patients unable to take oral medications (1:1 dose conversion); infuse over 30–60 minutes.
  3. Minimal drug interactions due to low protein binding and minimal CYP metabolism; good choice in complex polypharmacy patients.
  4. Monitor for dizziness and diplopia, especially with initiation; start at low doses and titrate slowly.

Clinical Pearls

  • Lacosamide's unique mechanism targeting slow Na+ channel inactivation means it works via a different molecular mechanism than phenytoin and carbamazepine, making combination with these agents pharmacodynamically complementary rather than redundant.
  • The IV formulation with 1:1 oral bioequivalence makes lacosamide practical for hospitalized patients who cannot take oral medications — an advantage over many other AEDs.

Safety Profile

Pregnancy use-with-caution
Lactation insufficient-data
Renal Adjustment Required
Hepatic Adjustment Required
TDM Not required

Concordance Terms

Cross-referenced clinical concepts — click any term to see all content where it appears.