lanthanum carbonate

Brand: Fosrenol

Prototype: sevelamer
Drug Class: phosphate binder
Drug Family: electrolyte agent
Subclass: non-calcium rare earth metal phosphate binder
Organ Systems: renal

Mechanism of Action

Lanthanum ions dissociate in the acidic gastric environment and bind dietary phosphate to form highly insoluble lanthanum phosphate complexes that are not absorbed and are excreted in feces; highly potent phosphate binding that is calcium- and aluminum-free.

dietary phosphate (GI lumen)

Indications

  • hyperphosphatemia in end-stage renal disease (dialysis patients)

Contraindications

  • hypophosphatemia
  • bowel obstruction
  • hypersensitivity

Adverse Effects

Common

  • nausea
  • vomiting
  • abdominal pain
  • diarrhea
  • constipation

Serious

  • GI obstruction (rare)
  • hepatotoxicity (rare)
  • long-term tissue accumulation (bone, liver, GI mucosa) — significance uncertain

Pharmacokinetics (ADME)

Absorption systemic absorption is minimal (<0.002% of dose); lanthanum accumulates in bone and GI mucosa long-term
Distribution minimally absorbed lanthanum distributes to bone and reticuloendothelial system
Metabolism not hepatically metabolized
Excretion fecal (>99%) as lanthanum phosphate complexes
Half-life N/A for pharmacological action; systemic lanthanum has a very long half-life (years) in tissue
Onset phosphate reduction within weeks
Peak N/A
Duration requires consistent dosing with meals
Protein Binding N/A (GI lumen action)
Vd N/A

Drug Interactions

Drug / Agent Mechanism Severity
oral medications generally lanthanum may bind other oral medications in the GI lumen; separate administration by 2 hours moderate
fluoroquinolones and tetracyclines metal ion chelation reduces antibiotic absorption moderate

Nursing Considerations

  1. Administer chewable tablets with or immediately after meals; instruct patients to chew tablets thoroughly before swallowing — do not swallow whole.
  2. Monitor serum phosphate, calcium, and iPTH every 3 months; target serum phosphate 3.5-5.5 mg/dL in dialysis patients per KDIGO guidelines.
  3. Long-term tissue accumulation of lanthanum (in bone, liver, and GI mucosa) has been documented in post-marketing studies; while clinical significance remains unclear, counsel patients about this phenomenon and report any unusual symptoms.
  4. Separate lanthanum carbonate from other oral medications by at least 2 hours to minimize GI binding; this is particularly important for fluoroquinolones, tetracyclines, and other drugs affected by metal chelation.

Clinical Pearls

  • Lanthanum carbonate requires tablet chewing before swallowing — unlike sevelamer tablets — which affects patient adherence; palatability issues (chalky taste) are the most common reason for non-adherence.
  • Among all phosphate binders, lanthanum binds phosphate most potently across the widest pH range (effective in both acidic gastric and neutral intestinal environments), potentially allowing lower pill burden than calcium-based binders.

Safety Profile

Pregnancy avoid
Lactation avoid
Renal Adjustment Not required
Hepatic Adjustment Not required
TDM Not required

Concordance Terms

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