levalbuterol

Brand: Xopenex

Prototype: albuterol
Drug Class: beta-2 adrenergic agonist (SABA)
Drug Family: bronchodilator
Subclass: R-enantiomer of albuterol — short-acting beta-2 agonist
Organ Systems: respiratory

Mechanism of Action

R-enantiomer of racemic albuterol; selectively activates beta-2 adrenergic receptors in bronchial smooth muscle, stimulating adenylyl cyclase, increasing cAMP, and activating PKA to phosphorylate myosin light chain kinase, causing bronchial smooth muscle relaxation and bronchodilation.

beta-2 adrenergic receptors (bronchial smooth muscle)

Indications

  • acute bronchospasm (asthma, COPD)
  • prevention of exercise-induced bronchospasm

Contraindications

  • hypersensitivity to levalbuterol or albuterol

Adverse Effects

Common

  • tachycardia
  • tremor
  • nervousness
  • headache
  • palpitations

Serious

  • severe paradoxical bronchospasm
  • hypokalemia (with high doses)
  • lactic acidosis (rare, high doses in severe asthma)

Pharmacokinetics (ADME)

Absorption inhaled; systemic bioavailability approximately 10-20% via nebulization
Distribution Vd approximately 660 L; crosses placenta
Metabolism hepatic conjugation to inactive metabolites; some undergoes sulfoconjugation
Excretion renal (predominantly as metabolites)
Half-life approximately 3.3-4 hours
Onset 5-15 minutes
Peak 1-1.5 hours
Duration 5-8 hours
Protein Binding approximately 10%
Vd approximately 660 L

Drug Interactions

Drug / Agent Mechanism Severity
non-selective beta-blockers competitive antagonism reduces bronchodilatory effect and may precipitate severe bronchospasm major
MAOIs and tricyclic antidepressants potentiate cardiovascular effects of sympathomimetics moderate
loop and thiazide diuretics additive hypokalemia risk at high levalbuterol doses moderate

Nursing Considerations

  1. Administer via nebulizer; do not mix with other solutions unless compatibility is confirmed; unit-dose vials (0.31, 0.63, 1.25 mg) simplify dosing for pediatric patients.
  2. Monitor heart rate and oxygen saturation during and after nebulization; tachycardia and tremor are common; discontinue and notify provider if paradoxical bronchospasm occurs.
  3. Monitor serum potassium in patients receiving frequent doses or who are also on diuretics; high-dose beta-2 agonist therapy can cause clinically significant hypokalemia.
  4. The proposed clinical advantage of levalbuterol over racemic albuterol (fewer cardiovascular side effects) has not been consistently demonstrated in controlled trials; cost is significantly higher.

Clinical Pearls

  • Levalbuterol is the R-enantiomer of racemic albuterol; the S-enantiomer (formerly believed to be inactive) may have pro-inflammatory properties, which was the rationale for developing levalbuterol as a purer bronchodilator.
  • Clinical evidence has not consistently demonstrated a significant advantage of levalbuterol over racemic albuterol in efficacy or tolerability; most guidelines consider them therapeutically equivalent at equipotent doses.

Safety Profile

Pregnancy use-with-caution
Lactation use-with-caution
Renal Adjustment Not required
Hepatic Adjustment Not required
TDM Not required

Concordance Terms

Cross-referenced clinical concepts — click any term to see all content where it appears.