methimazole

Brand: Tapazole

Prototype Drug
Drug Class: antithyroid
Drug Family: antithyroid
Subclass: thionamide
Organ Systems: endocrine

Mechanism of Action

Inhibits thyroid peroxidase, blocking oxidation of iodide to iodine and organification (coupling of iodine to tyrosine residues) — thereby preventing thyroid hormone (T3, T4) synthesis; does NOT affect existing thyroid hormone stores (delayed onset of effect).

thyroid peroxidase

Indications

  • hyperthyroidism (Graves' disease — first-line antithyroid therapy)
  • preparation for thyroid surgery or radioiodine therapy
  • thyroid storm (high doses)

Contraindications

  • first trimester of pregnancy (use PTU instead — methimazole causes aplasia cutis and choanal atresia)
  • hypersensitivity to methimazole

Adverse Effects

Common

  • rash
  • pruritus
  • nausea
  • arthralgias
  • mild leukopenia

Serious

  • agranulocytosis (0.1–0.5% — most dangerous; usually within first 3 months)
  • hepatotoxicity (cholestatic jaundice)
  • hypothyroidism (with excessive dosing)
  • ANCA-positive vasculitis (rare, usually with PTU)
  • teratogenicity (aplasia cutis with first-trimester exposure)

Pharmacokinetics (ADME)

Absorption Rapid and complete oral absorption
Distribution Concentrates in thyroid gland; protein binding minimal
Metabolism Hepatic
Excretion Renal
Half-life 4–6 hours; but biological effect lasts much longer (dosing every 8–24 hours depending on dose)
Onset 1–2 weeks for clinical effect (existing thyroid hormone must be consumed)
Peak 0.5–1 hour
Duration 8–24 hours
Protein Binding minimal
Vd ~0.6 L/kg

Drug Interactions

Drug / Agent Mechanism Severity
warfarin hyperthyroid state increases warfarin metabolism; normalization with methimazole increases warfarin effect — INR may rise significantly major
beta-blockers hyperthyroid state increases beta-blocker clearance; levels normalize as euthyroidism is achieved moderate
digoxin hypothyroidism increases digoxin sensitivity; monitor digoxin levels moderate

Nursing Considerations

  1. Instruct patient to report sore throat, fever, or mouth sores immediately — these are signs of agranulocytosis; obtain CBC with differential urgently; hold methimazole pending results
  2. Monitor CBC and LFTs at baseline and periodically; CBC before initiation establishes baseline WBC
  3. Monitor thyroid function tests (TSH, free T4) every 4–6 weeks during initial titration; TSH may remain suppressed for months after achieving euthyroidism — use free T4 to guide dose adjustments initially
  4. Use PTU (not methimazole) in first trimester of pregnancy; switch to methimazole after first trimester (PTU carries hepatotoxicity risk)

Clinical Pearls

  • Methimazole is preferred over PTU except in first-trimester pregnancy because it has once-daily dosing (vs PTU TID), lower hepatotoxicity risk, and equivalent efficacy — the ATA guidelines recommend methimazole as first-line for Graves' disease
  • Agranulocytosis from methimazole is idiosyncratic (not dose-dependent), can develop suddenly, and is potentially life-threatening — patients must be educated about this risk and instructed to present immediately for CBC if febrile or ill

Safety Profile

Pregnancy use-with-caution
Lactation use-with-caution
Renal Adjustment Not required
Hepatic Adjustment Required
TDM Not required