mirabegron

Brand: Myrbetriq

Prototype Drug
Drug Class: beta-3 adrenergic agonist
Drug Family: GU agent
Subclass: selective beta-3 agonist for overactive bladder
Organ Systems: reproductive

Mechanism of Action

Selectively activates beta-3 adrenergic receptors in detrusor smooth muscle, stimulating adenylyl cyclase and increasing cAMP, which relaxes the bladder muscle during the filling phase and increases bladder capacity. Unlike antimuscarinics, this mechanism does not affect bladder emptying or cause dry mouth/constipation.

beta-3 adrenergic receptor (detrusor smooth muscle)

Indications

  • overactive bladder (urge incontinence, urgency, urinary frequency)
  • neurogenic detrusor overactivity

Contraindications

  • severe uncontrolled hypertension
  • hypersensitivity

Adverse Effects

Common

  • hypertension (increased blood pressure)
  • nasopharyngitis
  • urinary tract infections
  • headache

Serious

  • urinary retention (in patients with bladder outlet obstruction)
  • tachycardia (QTc prolongation reported at supratherapeutic doses)

Pharmacokinetics (ADME)

Absorption oral bioavailability ~29–35%
Distribution 71% protein bound
Metabolism multiple pathways: CYP3A4, MAO, COMT, amide hydrolysis
Excretion renal (55%) and fecal (34%)
Half-life 50 hours
Onset weeks for full effect
Peak 3–4 hours
Duration 24 hours
Protein Binding 71%
Vd 1670 L

Drug Interactions

Drug / Agent Mechanism Severity
digoxin mirabegron inhibits P-gp and CYP2D6; increases digoxin levels; monitor digoxin major
metoprolol CYP2D6 inhibition increases metoprolol levels moderate

Nursing Considerations

  1. Monitor blood pressure before initiation and periodically; mirabegron raises blood pressure and is contraindicated in severe uncontrolled hypertension.
  2. Unlike antimuscarinics, mirabegron has no anticholinergic side effects — no dry mouth, constipation, or cognitive effects; preferred in elderly patients.
  3. Ensure patients have adequate bladder emptying ability; use with caution in patients with BPH or partial outlet obstruction (urinary retention risk).
  4. Educate patients to take consistently with or without food; full effect may take several weeks.

Clinical Pearls

  • Mirabegron is the first-in-class beta-3 agonist for overactive bladder, providing an alternative mechanism to antimuscarinics (oxybutynin, tolterodine) without the troublesome anticholinergic effects that often limit adherence.
  • The Beers criteria recommend antimuscarinics for OAB be avoided in elderly patients due to cognitive effects; mirabegron is increasingly preferred as the first-line OAB treatment in older adults.

Safety Profile

Pregnancy avoid
Lactation avoid
Renal Adjustment Required
Hepatic Adjustment Required
TDM Not required

Concordance Terms

Cross-referenced clinical concepts — click any term to see all content where it appears.