vonoprazan

Brand: Voquezna

Prototype Drug
Drug Class: potassium-competitive acid blocker (P-CAB)
Drug Family: GI agent
Subclass: potassium-competitive acid blocker
Organ Systems: gastrointestinal

Mechanism of Action

Competitively and reversibly inhibits gastric H+/K+-ATPase by competing with potassium ions at the enzyme's potassium-binding site, achieving rapid, potent, and sustained acid suppression without requiring an acidic environment for activation.

H+/K+-ATPase (proton pump)

Indications

  • erosive esophagitis
  • H. pylori eradication (combination therapy)
  • GERD

Contraindications

  • hypersensitivity to vonoprazan
  • concurrent rilpivirine use
  • concurrent atazanavir use

Adverse Effects

Common

  • diarrhea
  • nausea
  • abdominal pain
  • upper respiratory tract infection

Serious

  • Clostridium difficile infection
  • hypomagnesemia
  • acute interstitial nephritis

Pharmacokinetics (ADME)

Absorption oral bioavailability ~83%; not significantly affected by food
Distribution 80% protein bound
Metabolism hepatic via CYP3A4 (major) and CYP2B6/2C19 (minor)
Excretion renal and fecal
Half-life 9 hours
Onset 0.5–1 hour
Peak 1.5–2 hours
Duration 24+ hours
Protein Binding 80%
Vd moderate

Drug Interactions

Drug / Agent Mechanism Severity
atazanavir markedly reduced atazanavir absorption due to elevated gastric pH major
rilpivirine reduced rilpivirine absorption; contraindicated combination major
strong CYP3A4 inhibitors increased vonoprazan exposure moderate

Nursing Considerations

  1. Vonoprazan achieves more rapid and consistent acid suppression than PPIs; educate patients it works differently from traditional PPIs.
  2. Does not require activation by acid, so timing relative to meals is less critical than for PPIs.
  3. Monitor for C. difficile infection, particularly in hospitalized patients or those on antibiotics.
  4. Assess for concurrent use of HIV medications (rilpivirine, atazanavir) as these are absolute contraindications.

Clinical Pearls

  • Unlike PPIs, vonoprazan acts via reversible potassium-competitive inhibition and does not require acid activation, resulting in a faster onset and more complete acid suppression.
  • FDA-approved in combination with amoxicillin (dual therapy) or amoxicillin plus clarithromycin (triple therapy) for H. pylori eradication.

Safety Profile

Pregnancy avoid
Lactation avoid
Renal Adjustment Required
Hepatic Adjustment Required
TDM Not required
Guideline Update pending

Concordance Terms

Cross-referenced clinical concepts — click any term to see all content where it appears.