vortioxetine

Brand: Trintellix

⚠ BBW Prototype Drug
Drug Class: multimodal antidepressant
Drug Family: antidepressant
Subclass: serotonin modulator and stimulator
Organ Systems: cns

Mechanism of Action

Combines SERT inhibition with direct modulation of multiple serotonin receptor subtypes: agonism at 5-HT1A (autoreceptors, anxiolytic), partial agonism at 5-HT1B, and antagonism at 5-HT3 and 5-HT7 (reduces nausea and may enhance cognitive function). This multimodal activity distinguishes it from conventional SSRIs.

SERT (serotonin transporter)5-HT1A receptor (agonist)5-HT1B receptor (partial agonist)5-HT3 receptor (antagonist)5-HT7 receptor (antagonist)

Indications

  • major depressive disorder

Contraindications

  • concurrent MAOI use

Adverse Effects

Common

  • nausea
  • diarrhea
  • dry mouth
  • dizziness
  • constipation

Serious

  • serotonin syndrome
  • suicidal ideation
  • abnormal bleeding
  • hyponatremia

Pharmacokinetics (ADME)

Absorption well absorbed orally; bioavailability ~75%; not significantly affected by food
Distribution protein binding ~98%; Vd ~2600 L
Metabolism primarily via CYP2D6 (major) with minor contributions from CYP3A4, 2A6, 2B6, 2C8, 2C9, 2C19
Excretion renal (~59%) and fecal (~26%); primarily as inactive metabolites
Half-life 66 hours
Onset 2-4 weeks for full antidepressant effect
Peak 7-11 hours
Duration 24 hours
Protein Binding 98%
Vd 2600 L

Drug Interactions

Drug / Agent Mechanism Severity
MAOIs serotonin syndrome contraindicated
CYP2D6 inhibitors (bupropion, paroxetine) increases vortioxetine exposure; reduce dose by half major
CYP inducers (rifampin) decreases vortioxetine levels; increase dose up to 3x moderate

Nursing Considerations

  1. Nausea is the most common adverse effect and is dose-dependent; starting at 5 mg and titrating up over 2-4 weeks can reduce nausea during initiation.
  2. CYP2D6 inhibitors (bupropion, paroxetine, fluoxetine) significantly increase vortioxetine levels — review combination therapy and consider dose reduction to 10 mg/day.
  3. Vortioxetine has been studied for cognitive function in depression, showing improvements in processing speed and executive function; this may be a differentiating factor for patients with prominent cognitive symptoms.
  4. Unlike SSRIs, sexual dysfunction appears to be lower with vortioxetine (rates similar to placebo in trials); this may be a consideration for patients with prior SSRI-induced sexual side effects.

Clinical Pearls

  • Vortioxetine's clinical differentiation from SSRIs is its potential pro-cognitive effect; it is one of the few antidepressants with RCT data showing improvement in cognitive domains (memory, attention, processing speed) beyond mood symptoms.
  • Its 66-hour half-life allows for some flexibility in missed doses without the precipitous withdrawal symptoms seen with short-half-life drugs like paroxetine.

Safety Profile

Pregnancy use-with-caution
Lactation use-with-caution
Renal Adjustment Not required
Hepatic Adjustment Not required
TDM Not required

Concordance Terms

Cross-referenced clinical concepts — click any term to see all content where it appears.