zafirlukast

Brand: Accolate

Prototype: montelukast
Drug Class: leukotriene receptor antagonist (LTRA)
Drug Family: leukotriene modifier
Subclass: oral competitive cysteinyl leukotriene receptor antagonist
Organ Systems: respiratory

Mechanism of Action

Competitive antagonist at CysLT1 receptors in airway smooth muscle and inflammatory cells; blocks leukotriene D4 and E4 (products of the 5-lipoxygenase pathway acting on arachidonic acid), preventing bronchoconstriction, airway edema, increased mucus secretion, and eosinophil recruitment.

cysteinyl leukotriene CysLT1 receptor

Indications

  • persistent asthma (maintenance therapy, adults and children 5 years and older)
  • prophylaxis of exercise-induced bronchospasm

Contraindications

  • hepatic impairment (metabolized by CYP2C9; increased hepatotoxicity risk)
  • hypersensitivity

Adverse Effects

Common

  • headache
  • nausea
  • diarrhea
  • infection

Serious

  • hepatotoxicity (rare but potentially fatal — cases reported)
  • Churg-Strauss syndrome (eosinophilic vasculitis — with corticosteroid tapering)
  • neuropsychiatric events (rare: insomnia, behavioral changes)

Pharmacokinetics (ADME)

Absorption oral; reduced by food (40% reduction in Cmax and AUC) — take on empty stomach
Distribution Vd approximately 70 L; >99% protein bound
Metabolism extensive hepatic CYP2C9 metabolism to inactive hydroxylated metabolites; also a CYP2C9 inhibitor
Excretion fecal (~89%) and renal (~10%)
Half-life approximately 10 hours
Onset days for optimal anti-inflammatory effect
Peak 3 hours
Duration 12 hours (requires twice-daily dosing — unlike montelukast)
Protein Binding >99%
Vd approximately 70 L

Drug Interactions

Drug / Agent Mechanism Severity
warfarin zafirlukast inhibits CYP2C9; increases warfarin plasma levels and anticoagulant effect — monitor INR major
aspirin increases zafirlukast plasma levels by approximately 45% moderate
erythromycin and theophylline decrease zafirlukast plasma levels; may reduce efficacy moderate

Nursing Considerations

  1. Administer on an empty stomach (1 hour before or 2 hours after meals) as food significantly reduces drug absorption; this twice-daily fasting requirement is a key adherence challenge compared with once-daily montelukast.
  2. Monitor liver enzymes at baseline and if symptoms of hepatotoxicity develop (jaundice, right upper quadrant pain, fatigue); discontinue if hepatotoxicity is confirmed.
  3. Closely monitor warfarin INR when zafirlukast is initiated or discontinued; CYP2C9 inhibition raises warfarin levels and may require warfarin dose reduction.
  4. Educate patients about neuropsychiatric risks (insomnia, anxiety, behavioral changes) — though primarily associated with montelukast, any LTRA can potentially cause these effects; report changes promptly.

Clinical Pearls

  • Zafirlukast requires twice-daily fasting administration, placing it at an adherence disadvantage compared with once-daily montelukast; most prescribers prefer montelukast for long-term LTRA therapy, reserving zafirlukast for specific cases or formulary requirements.
  • The CYP2C9 inhibitory effect of zafirlukast on warfarin metabolism is clinically significant and has caused serious bleeding events; the warfarin interaction is a key differentiator from montelukast and a priority patient safety consideration.

Safety Profile

Pregnancy generally-safe
Lactation use-with-caution
Renal Adjustment Not required
Hepatic Adjustment Required
TDM Not required